Base color
Model & settings
Image simulation
Upload JPG/PNG (max 1200×1200). Simulated canvas
next to original.
Research notes
Cortical visual impairment (CVI) results from damage to the visual cortex
(V1–V4) or posterior visual processing pathways, most commonly from periventricular
leukomalacia (PVL) or hypoxic-ischemic encephalopathy (HIE). Visual complexity reduction
simulates the difficulty CVI patients experience parsing cluttered visual scenes. Color
preference mode emphasizes red/yellow wavelengths preferred by many CVI individuals. Latency
mode applies temporal blur to approximate delayed visual processing response.
Swatches — Reference vs Cortical Processing Loss
Reference
HEX: — • RGB: — • xy: —
CVI Sim
HEX: — • RGB: — • xy: —
ΔE (CIE76)
—
ΔE (CIEDE2000)
—
Deep preview
Reference
Simulated
Chromaticity (CIE xy)
Achromatic axis (to D65)
Image simulation
Original
Cortical visual impairment simulated
Multi-condition comparison
5
Clinical notes: Bouma ×1.0 reproduces the canonical critical spacing
(half eccentricity) measured in typical CVI subjects. Increasing the factor simulates
more severe periventricular white-matter damage extending beyond V1 into dorsal stream.
High contrast sensitivity exponent (>1.5) models profound low-contrast processing
failure characteristic of occipital white-matter injury.
Model assumptions & limits
- Crowding blur is a spatial approximation using a Gaussian kernel scaled by the Bouma factor; true cortical crowding involves nonlinear lateral inhibition in V1 and extrastriate cortex.
- Contrast reduction is applied as a global power function; real CVI contrast deficit is spatial-frequency and eccentricity dependent.
- ΔE metrics compare swatch color only and do not model the full perceptual complexity reduction experienced by CVI individuals in dynamic scenes.